Biology is designed for multi-semester biology courses for science majors. It is grounded on an evolutionary basis and includes exciting features that highlight careers in the biological sciences and everyday applications of the concepts at hand. To meet the needs of today’s instructors and students, some content has been strategically condensed while maintaining the overall scope and coverage of traditional texts for this course. Instructors can customize the book, adapting it to the approach that works best in their classroom. Biology also includes an innovative art program that incorporates critical thinking and clicker questions to help students understand—and apply—key concepts.

By the end of this section, you will be able to:Discuss how fertilization occursExplain how the embryo forms from the zygoteDiscuss the role of cleavage and gastrulation in animal development

By the end of this section, you will be able to:List the features that distinguish the kingdom Animalia from other kingdomsExplain the processes of animal reproduction and embryonic developmentDescribe the roles that Hox genes play in development

Hank describes mitosis and cytokinesis - the series of processes our cells go through to divide into two identical copies.

Chapters:
1. Mitosis
2. Interphase
a) Chromatin
b) Centrosomes
3) Prophase
a) Chromosomes
b) Chromatid
c) Microtubules
4) Metaphase
a) Motor Proteins
5) Biolography
6) Anaphase
7) Telophase
8) Cleavage
9) Cytokinesis

This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:

"Alzheimer’s disease and glioblastoma are the most common and devastating diseases in neurology and neurosurgery departments. In glioblastoma, the Alzheimer’s disease-related protein Presenilin1 limits cell proliferation by inhibiting the Wnt/β-catenin pathway. However, the function of this protein and the underlying mechanism are unclear. To learn more, researchers recently examined Presenilin1’s effects on glioblastoma in vitro and in vivo. Presenilin1 repressed glioblastoma cell migration, invasion, and mesenchymal transition in vitro. Interestingly, its expression was positively correlated with that of Sortilin, a pro-invasion molecule in glioma. Presenilin1 interacted with Sortilin at the transmembrane domain and inhibited its expression via cleavage, enabling β-catenin phosphorylation and repressing invasion in glioblastoma cells. In mouse subcutaneous and intracranial transplantation models, Presenilin1 stimulation dramatically reduced glioblastoma invasion and mesenchymal transition..."

The rest of the transcript, along with a link to the research itself, is available on the resource itself.