Biology is designed for multi-semester biology courses for science majors. It is …
Biology is designed for multi-semester biology courses for science majors. It is grounded on an evolutionary basis and includes exciting features that highlight careers in the biological sciences and everyday applications of the concepts at hand. To meet the needs of today’s instructors and students, some content has been strategically condensed while maintaining the overall scope and coverage of traditional texts for this course. Instructors can customize the book, adapting it to the approach that works best in their classroom. Biology also includes an innovative art program that incorporates critical thinking and clicker questions to help students understand—and apply—key concepts.
By the end of this section, you will be able to:Explain how …
By the end of this section, you will be able to:Explain how hormones regulate the excretory systemDiscuss the role of hormones in the reproductive systemDescribe how hormones regulate metabolismExplain the role of hormones in different diseases
This resource is a video abstract of a research paper created by …
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:
"In lactating mammals, the mammary glands are responsible for milk production and secretion. Low energy levels in mammary gland cells are known to reduce milk synthesis by activating AMPK, an energy-sensing protein. However, the exact mechanism by which energy status affects lactation efficiency isn’t clear. To learn more, researchers recently examined AMPK’s role in milk synthesis in mouse mammary epithelial cells. Energy (glucose) deficiency indeed activated AMPK, which reduced milk fat and protein synthesis in the cells. AMPK inhibited milk production partly by inhibiting the classic mTORC1 signaling pathway, but it primarily reduced milk protein synthesis by blocking the signaling pathway of the lactation hormone prolactin. Specifically, AMPK triggered degradation of the prolactin receptor (PrlR) in lysosomes to halt prolactin signaling..."
The rest of the transcript, along with a link to the research itself, is available on the resource itself.
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