Biology is designed for multi-semester biology courses for science majors. It is …
Biology is designed for multi-semester biology courses for science majors. It is grounded on an evolutionary basis and includes exciting features that highlight careers in the biological sciences and everyday applications of the concepts at hand. To meet the needs of today’s instructors and students, some content has been strategically condensed while maintaining the overall scope and coverage of traditional texts for this course. Instructors can customize the book, adapting it to the approach that works best in their classroom. Biology also includes an innovative art program that incorporates critical thinking and clicker questions to help students understand—and apply—key concepts.
By the end of this section, you will be able to:Describe human …
By the end of this section, you will be able to:Describe human male and female reproductive anatomiesDiscuss the human sexual responseDescribe spermatogenesis and oogenesis and discuss their differences and similarities
This resource is a video abstract of a research paper created by …
This resource is a video abstract of a research paper created by Research Square on behalf of its authors. It provides a synopsis that's easy to understand, and can be used to introduce the topics it covers to students, researchers, and the general public. The video's transcript is also provided in full, with a portion provided below for preview:
"Male infertility due to toxin exposure is a serious health issue worldwide. However, the metabolic link between toxin exposure and testicular dysfunction is unclear. To learn more, a recent study examined the metabolic disruption associated with triptolide (TP)-induced testicular injury in mice. TP caused spermine (SP) deficiency by disrupting polyamine biosynthesis and uptake in the testes and by perturbing the gut microbiota. Exogenous SP supplementation reversed the TP-induced testicular dysfunction by increasing the expression of early and late spermatogenesis-related genes and by attenuating TP-induced decreases in offspring numbers. SP’s protective effect was found to be largely dependent on upregulation of HSP70 proteins in vivo and in vitro..."
The rest of the transcript, along with a link to the research itself, is available on the resource itself.
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